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Tseng K.-Y. (ed.) Cortico-Subcortical Dynamics in Parkinson's Disease

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Tseng K.-Y. (ed.) Cortico-Subcortical Dynamics in Parkinson's Disease
New York: Humana Press, 2009. — 440 p.
Cortico-subcortical dynamics in Parkinson’s disease aims to integrate key pathophysiological aspects underlying Parkinson’s disease. The volume offers a broad spectrum of opinions on how chronic dopamine depletion affects cortico-subcortical dynamics, especially how disruptions of the non-dopaminergic systems resulting from chronic dopaminergic degeneration could lead to the functional alterations observed in parkinsonism.
Leading Toward a Unified Cortico-basal Ganglia Functional Model
Modeling Parkinson’s Disease: 50 Years Later
Phasic Dopaminergic Signaling: Implications for Parkinson’s Disease
Striatal Dendritic Adaptations in Parkinson’s Disease Models
Diversity of Up-State Voltage Transitions During Different Network States
The Corticostriatal Pathway in Parkinson’s Disease
Cholinergic Interneuron and Parkinsonism
Basal Ganglia Network Synchronization in Animal Models of Parkinson’s Disease
Converging into a Unified Model of Parkinson’s Disease Pathophysiology
The Corticostriatal Transmission in Parkinsonian Animals: In Vivo Studies
Striatal Nitric Oxide–cGMP Signaling in an Animal Model of Parkinson’s Disease
Dopamine–Endocannabinoid Interactions in Parkinson’s Disease
Glutamate Plasticity in an Animal Model of Parkinson’s Disease
Neuromodulation and Neurodynamics of Striatal Inhibitory Networks: Implications for Parkinson’s Disease
Dopaminergic Modulation of Corticostriatal Interactions and Implications for Parkinson’s Disease
Pathogenesis of Oxidative Stress and the Destructive Cycle in the Substantia Nigra in Parkinson’s Disease
Regulation of G-Protein-Coupled Receptor (GPCR) Trafficking in the Striatum in Parkinson’s Disease
Atypical Parkinsonism in the French West Indies: The Plant Toxin Annonacin as a Potential Etiological Factor
Cognitive Deficits in Parkinson’s Disease
Dopamine Replacement Therapy in Parkinson’s Disease: Past, Present and Future
Molecular, Cellular and Electrophysiological Changes Triggered by High-Frequency Stimulation of the Subthalamic Nucleus in Animal Models of Parkinson’s Disease
Surgical Strategies for Parkinson’s Disease Based on Animal Model Data: GPi and STN Inactivation on Various Aspects of Behavior (Motor, Cognitive and Motivational Processes)
Antidromic Cortical Activity as the Source of Therapeutic Actions of Deep Brain Stimulation
Cell-Based Replacement Therapies for Parkinson’s Disease
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